Ear, Nose & Throat
Tonsillitis, epistaxis, ear infections & airway emergencies.
Use as a study guide only. These notes are part of a free open-access medical education (FOAMed) project and may contain errors or outdated information. Always verify against current guidelines (e.g. eTG, RACGP, local health district policies) and reputable sources before applying anything to patient care. See the full disclaimer.
Clinical pearls & learnings
Tips and tricks collected over the years — a living list that grows with every rotation. Use as a study guide only and check current guidelines before acting on anything.
Tonsillitis & tonsillectomy
The sore throat that isn't quinsy
- Most acute tonsillitis is viral, not bacterial — group A strep is the main bacterial cause worth treating, but it's the minority.
- Most sore throats are simple tonsillitis, not a peritonsillar abscess (quinsy) — trismus, uvular deviation and a muffled "hot potato" voice point towards quinsy and need same-day ENT assessment.
- Avoid amoxicillin/ampicillin where infectious mononucleosis (EBV) is possible — it classically triggers a widespread maculopapular rash in glandular fever.
Quinsy — recognising and managing a peritonsillar abscess
- Exam findings: soft palate fullness with loss of the peritonsillar arch, uvula deviated away from the affected side, trismus, and a muffled "hot potato" voice.
- Key differential: peritonsillar cellulitis — diffuse inflammation without a drainable pus collection, which can progress to a true abscess if untreated.
- Diagnosis is clinical ± bedside intraoral or transcervical ultrasound; CT is not routinely needed for a typical presentation — reserve it for atypical features, suspected deep neck space extension, or when trismus prevents an adequate exam.
- Dexamethasone 8 mg IV STAT + IV antibiotics (amoxicillin-clavulanate covers the typical polymicrobial oral flora)
- Topical anaesthesia — co-phenylcaine (lidocaine) spray
- Local anaesthetic ± adrenaline infiltration
- Needle aspiration first — often diagnostic and therapeutic on its own
- If aspiration is inadequate: incision and drainage with blunt dissection, exploring the cavity to break down loculations
- Post-procedure: dilute (3%) hydrogen peroxide gargles
Management
Refer to ENT for the procedure itself — a tongue depressor, good lighting and patient cooperation (trismus can make this difficult) are needed throughout.
- Practical procedural points: written consent, a 20mL syringe for aspiration, and drain with a capped/guarded 18-gauge needle — capping limits insertion depth and helps avoid the carotid sheath, which sits close by.
- Pre-procedure adjuncts some operators use: co-phenylcaine spray, glycopyrrolate to dry secretions and improve the view, and nebulised adrenaline with lidocaine.
- CT neck specifically if retropharyngeal or other deep neck space extension is suspected (see the diagnosis note above).
- After drainage: oral augmentin for 7 days, plus warm salt water gargles for comfort and hygiene.
Consider Lemierre syndrome if a sore throat isn't settling and the patient develops systemic sepsis — septic thrombophlebitis of the internal jugular vein (classically Fusobacterium necrophorum), presenting with ipsilateral neck fullness/tenderness along the vein and septic emboli, often to the lungs.
When tonsillectomy is actually indicated
- Obstructive sleep apnoea attributable to tonsillar hypertrophy.
- Paradise criteria for recurrent tonsillitis: ≥7 episodes in 1 year, or ≥5/year for 2 years, or ≥3/year for 3 years.
- Recurrent peritonsillar abscess (typically ≥2 episodes).
- Suspected malignancy.
Pain classically flares again around day 5–8 post-tonsillectomy as the fibrinous membrane over the tonsillar bed separates — expected, and worth warning patients about. But this is also the highest-risk window for secondary haemorrhage, so any actual bleeding at this time is never "just normal healing" and needs urgent assessment.
Post-tonsillectomy bleed — treat as an airway emergency
Primary bleeds occur within 24 hours; secondary bleeds (the more common presentation to ED) occur 5–10 days post-op. Involve ENT and anaesthetics early — the danger is airway compromise and aspiration, not just blood loss.
- Sit the patient up and lean forward to protect the airway from pooling/aspirated blood.
- Two large-bore IV cannulae, IV fluids, and bloods including FBC, coagulation studies and a group & hold (crossmatch if bleeding is significant).
- TXA IV as an adjunct while arranging definitive management.
- Temporising measures for visible ooze: co-phenylcaine (lidocaine + phenylephrine) spray, or adrenaline-soaked gauze held to the tonsillar bed.
Do not suction a clot sitting in the tonsillar bed — dislodging it can precipitate torrential bleeding.
Nasal fractures & epistaxis
Nasal fractures
- Largely a clinical diagnosis — deformity, swelling and epistaxis after trauma. Plain X-ray of the nasal bones rarely changes management and isn't routinely needed.
- Before treating the fracture itself, actively exclude: septal haematoma (needs urgent drainage — untreated it can cause cartilage necrosis and a saddle-nose deformity), CSF rhinorrhoea, and other serious head injury.
- Any open wound over a nasal fracture needs antibiotic cover. Cat bites specifically carry a high risk of Pasteurella multocida and are treated with amoxicillin-clavulanate.
Closed reduction is ideally performed once swelling has settled but before the bones fixate — commonly around day 5–10 post-injury in adults (children heal faster and are usually reduced sooner).
Epistaxis — first aid through to escalation
- History: anticoagulant/antiplatelet use, recent nasal surgery, trauma, hypertension. Review anticoagulation rather than reflexively ceasing it — weigh bleeding risk against the patient's thromboembolic risk.
- First aid: sit upright, lean forward (stops swallowed blood), firm continuous compression of the soft cartilaginous lower part of the nose for at least 15–20 minutes.
- Adjuncts: topical vasoconstrictor (oxymetazoline), TXA- or adrenaline-soaked gauze/pledgets, silver nitrate cautery for a visible anterior bleeding point (classically Little's area).
- If not controlled: nasal tampons or a balloon device (e.g. RapidRhino). Avoid packing both sides at once where possible — bilateral packing raises the risk of septal pressure necrosis.
Hypertension often accompanies epistaxis but is rarely the primary cause. Control the bleeding and the patient's pain/anxiety first. Reserve IV antihypertensives for a genuine hypertensive emergency with end-organ damage — not routine epistaxis; a persistently elevated BP once bleeding is controlled is usually managed with cautious oral agents.
A posterior bleed — anterior measures fail, or bleeding is visible tracking down the oropharynx — needs ENT referral and usually admission.
Ear infections
Otitis externa
- Swab if severe, recurrent, or not responding to first-line treatment.
- Aural microtoileting (gentle irrigation or microsuction) clears debris and improves topical drug penetration.
- Keep the ear dry — no swimming, use a plug when showering.
- Topical ciprofloxacin drops ± an ear wick if the canal is swollen shut.
- Topical antifungals for confirmed or suspected fungal otitis externa (otomycosis) — use a non-ototoxic formulation if there's any possibility of a perforated tympanic membrane.
Otitis media
Usually follows a viral upper respiratory tract infection causing eustachian tube dysfunction — most common in young children. Ear and jaw/TMJ pain can refer in either direction via shared sensory innervation, so a normal-looking ear with ongoing pain should prompt examination of the teeth and TMJ for a referred cause.
- Analgesia is first-line regardless of whether antibiotics are given.
- Consider antibiotics for: age under 2 years, immunocompromise, bilateral disease in a young child, perforated TM/otorrhoea, systemic illness, or symptoms not improving after 48–72 hours of watchful waiting.
- Amoxicillin is first-line; use amoxicillin-clavulanate if a beta-lactam was given in the last 30 days, there's been treatment failure, or there's a history of recurrent AOM.
- Complications: conductive hearing loss, otitis media with effusion (OME), mastoiditis (boggy, tender postauricular swelling with a protruding ear — can cause facial nerve palsy via the nerve's course through the temporal bone), and rare intracranial spread (meningitis, epidural abscess).
- Suspected mastoiditis: CT petrous bones to define extent, start IV antibiotics and involve ENT urgently.
- OME usually resolves within 3 months; persistent effusion beyond that — especially bilateral, with hearing loss — warrants ENT referral to consider grommets.
Neck swelling
Working through a neck lump
| Lump | Distinguishing features |
|---|---|
| Branchial cyst | Anterior to sternocleidomastoid; classically young adults, may enlarge with a viral URTI |
| Thyroglossal cyst | Midline; moves upward on tongue protrusion or swallowing (tethered to the foramen caecum) — the most common congenital neck mass in children |
| Abscess | Painful, erythematous, fluctuant |
| Reactive/enlarged lymph node | Skin moves freely over it; usually mobile and often tender if reactive |
| Epidermoid (sebaceous) cyst | Attached to the overlying skin — skin does NOT move over it, often with a visible central punctum |
| Lipoma | Painless, soft, rubbery, smooth and slow-growing |
A persistent, unexplained neck lump in an adult (especially if firm, fixed, or lasting more than 3 weeks) needs urgent referral to exclude malignancy — don't wait and review.
The suspicious lymph node
- A firm, fixed (rather than mobile) node raises concern for malignant infiltration.
- In an adult with risk factors (smoking, alcohol), the most common cause of a malignant cervical node is metastatic squamous cell carcinoma from the upper aerodigestive tract (oral cavity, oropharynx, larynx, hypopharynx) — work-up for an unknown primary includes ENT examination with nasoendoscopy, fine-needle or core biopsy of the node, and cross-sectional imaging ± panendoscopy.
- If a thyroid primary is suspected, note that papillary thyroid carcinoma (around 85% of thyroid cancers) is well known for presenting as a lateral neck node metastasis even when the primary tumour in the thyroid itself is small or occult.
Salivary gland stones
The submandibular gland is the most common site for salivary duct stones (sialolithiasis) — its saliva is more viscous and alkaline, and Wharton's duct runs an uphill course with a narrower distal opening, all of which favour stone formation.
Foreign bodies
Ear, nose and oesophagus
- Live insect in the ear canal: instil oil or topical lignocaine to kill/immobilise it before attempting removal.
- Consider procedural sedation (e.g. ketamine) for a distressed child needing a more invasive removal.
- Nasal FB removal options: direct visualisation with instruments, a suction catheter, positive-pressure techniques ("parent's kiss") in a cooperative child, or cyanoacrylate glue on a cotton bud touched to a smooth round object and withdrawn once adhered.
A suspected oesophageal food bolus impaction can sometimes be helped along with a trial of a fizzy drink (the "Coca-Cola technique") ± glucagon. This is for suspected food bolus only — never for a suspected sharp object or button battery — and shouldn't delay endoscopy if it fails or red flags are present (drooling, complete obstruction, chest pain).
Airway emergencies
The compromised upper airway
- Causes to consider: foreign body, anaphylaxis, malignancy, quinsy/odontogenic abscess, infective (e.g. epiglottitis) or inflammatory causes.
- For an infective/inflammatory threat: IV antibiotics, nebulised adrenaline as a temporising measure, IV dexamethasone, and urgent ENT/anaesthetic involvement — may need a controlled airway in theatre.
- Flexible nasoendoscopy by ENT is the key tool to directly visualise the airway when compromise is suspected — done cautiously with senior/anaesthetic support on standby, given the risk of precipitating complete obstruction.
A tracheostomy and a laryngectomy are not the same emergency. A laryngectomy is an end stoma — there is no connection between the mouth/nose and the trachea, so oral or nasal intubation is impossible; ventilation and intubation must go through the stoma only. A tracheostomy patient still has a patent upper airway that may be usable if the tube is blocked or dislodged. Confirm which one you're dealing with before you act.
